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Description
The dose-dependent activation of the FAK-paxillin pathway increases tendon fibroblast migration and survival under oxidative stress

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Additionally, they activate the AMPK pathway in liver cells, which promotes fatty acid oxidation and reduces lipid accumulation (lipogenesis), offering potential benefits for non-alcoholic fatty liver disease (NAFLD) (Zheng et al., 2024)
Clin Toxicol 1993;31:277-94 12
doi: 10.3389/fpubh.2024.1419361

Preclinical studies have indicated that both peptides are safe within recommended doses