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Hence, GSH depletion is not a relevant signaling mechanism after exposure to low, subtoxic ITC doses while treatment with cytotoxic ITC concentrations potentially triggers low GSH responsive genes ( Application of 14 C-labeled SF to animal cell lines revealed that extracellular SF was rapidly depleted in the medium and that intracellular enrichment of SF was driven by conjugation of SF to GSH

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Binding of glutamate changes the receptors conformation and allows influx of extracellular Na + and other cations, and an efflux of intracellular K + ions

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Sexually dimorphic and brain region-specific transporter adaptations in system xc null mice