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Obesity is a predominantly pro-inflammatory process, and the main mechanism is that in obesity, classically activated macrophages (M1) have higher concentrations than alternatively activated macrophages (M2), expressing pro-inflammatory cytokines, and activating pro-inflammatory pathways, such as the JNK and TNF-kB signaling pathways, which lead to low-grade chronic inflammation throughout the body (190)

J Biological Chemistry 261: 4986-4992

[DOI] [PubMed] [Google Scholar] Walter E, Kissel T, Raddatz P

Additionally, it was proved that thale cress seedlings of the SALK_060087 line, which carries a T-DNA insertion in the GLDH gene, do not develop beyond the cotyledon stage without adding AsA (Pineau et al., 2008)

Micrornas and other non-coding rnas in adipose tissue and obesity: emerging roles as biomarkers and therapeutic targets

300 Sato et al