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49 DavidsonR

These clinical features are classic for focal impaired awareness seizures originating in the medial temporal lobe, making the most appropriate target for intracranial electroencephalography electrodes region E

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Kaufman and Diamond, Figure 2 Interference With the Prion-Like Spread of -Syn Ongoing research seeks to identify disease-modifying agents for PD that will restrict templated conformation changes and transcellular propagation of pathological -syn

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ACR disrupts cellular redox homeostasis by increasing reactive oxygen species and nitric oxide synthase levels, leading to lipid peroxidation and the accumulation of malondialdehyde and nitric oxide
