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Non-genetic mechanisms have recently emerged as important drivers of anticancer drug resistance

We describe important clinical outcomes of several key molecules targeting components of the glutamatergic synapse and their applicability as RAADs

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Nickel agarose (left), streptavidin agarose (middle), and glutathione agarose (right), are used to detect interactions with his-tagged, strep-tagged, and GST-tagged target proteins, respectively