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Waqas M

In this review, we synthesize the current understanding of how dysregulated copper metabolism and cuproptosis drive the pathogenesis of major cardiovascular conditions, including myocardial ischemia/reperfusion (I/R) injury, anthracycline-induced cardiotoxicity, atherosclerosis, diabetic cardiomyopathy (DCM), and sepsis-induced cardiac dysfunction, through pathways such as mitochondrial dysfunction, oxidative stress, and inflammation

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Specifically, we examined changes in the tissue concentrations of both the reduced (GSH) and oxidized (GSSG) forms of glutathione, in addition to the ratio of GSH to GSSG

Rights and permissions About this article Cite this article Lee, J.K., Lee, HN., Agrawal, S

The more meaningful clinical concern is sourcing and compounding
