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S-glutathionylation of FABP5 inhibits LPS-induced inflammation in macrophages To gain insight into the changes in biological processes and pathways caused by FABP5 S-glutathionylation, differentially-expressed genes in macrophages were assessed by RNA-seq

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They are shining examples of how specific amino acid sequences can be leveraged to interact with biological systems in profoundly different ways

10.1016/j.redox.2023.102758 7 ArslanbaevaL.TosiG.RavazzoloM.SimonatoM.TucciF

Elevated IL-1 levels in AH patients are closely linked to inflammation

FIGURE 9 4 Discussion The pathophysiological mechanisms underlying ARD include stem and progenitor cell depletion within the basal and dermal layers, and fibrotic progression promoted by inflammatory mediators through multicellular interactions (Yarnold and Brotons, 2010