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Redox regulation of NLRP3 inflammasomes: ROS as trigger or effector

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If the ability to reduce GSSG to GSH is overwhelmed by a high burden of oxidative stress, GSSG can be exported out of cells or react with protein sulphydryl groups to prevent a major shift in the redox equilibrium [11], thereby depleting cellular GSH

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The slight fluctuations observed in the hydrogen bond counts across all complexes over time are likely attributed to the thermal motion

[1] Cell Line:human chronic myeloid leukemia K562 cells Concentration:10-40 g/mL Incubation Time:48 h Result:Induced dose-dependent S-phase cell cycle arrest, with S-phase population increasing from 48.99% (untreated) to 52.89% (10 g/mL), 56.05% (20 g/mL), and 70.04% (40 g/mL)