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Functional studies further demonstrate that gut microbiota may contribute to PDrelevant phenotypes

This hypothesis posits that with steatosis, the liver undergoes ER stress, oxidative stress and mitochondrial dysfunction, leading to an increase in oxidative metabolites, hepatocellular damage and secretion of a large number of inflammatory factors, which ultimately leads to further deterioration into liver disease, liver fibrosis, cirrhosis, inflammatory necrosis and other reactions (19,20)