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The oxidative stress-induced weakening of the fibrous cap covering the plaque, due to the degradation of extracellular matrix components by matrix metalloproteinases (MMPs), is a key factor in plaque instability [75]

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To further test whether NCOA4 383522 interacts with the LDS, we probed the ability of a peptide derived from ATG4B that is known to bind to the LDS (Skytte Rasmussen et al, 2017) to disrupt the binding of NCOA4 383522 to (GST-GATE16) 2 in BLI

also identified an equine NK(natural killer cell)-lysin which is specifically expressed in lymphocytes [37]