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Phenolic compounds are also responsible for the formation of anti-proliferative activity and provide this effect through various mechanisms such as inducing apoptosis, disrupting the cell cycle, altering caspase activities, increasing the number of cells in G 1 phase, and accelerating the expression of p53 and p21 87

36: 304451 Chaudhury C, Brooks CL, Carter DC, et al

Although the potential contribution of the TME is considered in mediating the observed in vivo effects, further research is needed to address the complex mechanisms altering these treatment responses

& Kurpisz, M

The ensuing release of DAMPs promotes DC recruitment and activation, initiating robust anti-tumor immune responses [276, 277]

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