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Analogous to the transient therapeutic effects of cardiac glycosides used in acute and chronic heart failure, short-term elevation of FGF21 acts primarily on adipose tissue to enhance glucose uptake and lipolysis, modulates hypothalamic energy balance and neuroendocrine output, and promotes hepatic fatty acid oxidation and ketogenesis (Szczepaska and Gietka-Czernel, 2022

Lead (Pb) and Neurodevelopment: A Review on Exposure and Biomarkers of Effect (BDNF, HDL) and Susceptibility

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Mitochondrial dysfunction inhibits insulin signaling pathway through the overproduction of ROS and interfering with oxidation of acetyl CoA, consequently resulting in increased lipid and diacylglycerol [1, 48, 66, 134]