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doi: 10.3390/ijms18020325

A recent study further demonstrated that WBP1-deficient CRC cells regained sensitivity to 5-FU following the restoration of mitochondrial function [65], reinforcing a causal link between mitochondrial homeostasis and drug responsiveness

Active mTOR attenuates the expression of autophagy genes by inhibiting the activation and nuclear localization of TFEB and other MiTF transcription factors through direct phosphorylation

Additionally, the neuronal activity of Nrf2 is limited by the high content of the Cullin 3 protein, contributing to the proteasome degradation of Nrf2 [18]

Superoxide radicals in the execution of cell death