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Excessive activation of the Ang II type 1 receptor induces NADPH oxidase, which is a major generator of ROS in vascular and CC, and elevated ROS level may diminish NO bioavailability

Examples include tryptases recruiting inflammatory cells into the lungs, neutrophil elastase inactivating tissue inhibitors of metalloproteinases, MMPs inactivating alpha-1 antitrypsin and macrophage inflammatory protein-1

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