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The molecular basis of renal tubular transport disorders

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LEC2 directly activates the expression of DRM2 , maintaining high CHH methylation levels to recruit the SUVH-SDJ-AHL complex, which results in chromatin decondensation and high expression levels of totipotency-regulating genes (Fig

Kaiser SJ, Mutters NT, Blessing B, Gnther F

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10.1111/jph.12808 10 AoyamaM.TadaM.YokooH.ItoT.MisawaT.DemizuY.et al (2024)
