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Therefore, we set out to investigate if, by using this type of analysis, the mechanisms of lung toxicity (especially pulmonary fibrosis) associated with the benchmark chemicals could be identified

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In one study employing 31 P-magnetic resonance spectroscopy in AD patients, the phosphomonoester levels decreased after ALC treatment, while there was a normalization of high-energy phosphate at baseline, with these changes being evident only in the dorsal prefrontal area [23]

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