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Among the key pathways implicated in adipose tissue (AT) inflammation are the toll-like receptor 4 (TLR4)/phosphatidylinositol- 3-kinase (PI3 K)/protein kinase B (Akt) pathway, the ER stress-induced unfolded protein response (UPR), and the IKK-NF-B inhibitory pathway are three metabolic pathways that are significant in the development of AT inflammation [94]

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