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Infants are introduced to exogenous L-carnitine and acetyl-carnitine through breast milk, although it is known that L-carnitine and acetyl-carnitine have no specific immediate effects in the breast-feeding child [23]

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However, another study confirmed that the mean TMAO levels did not change during the lifestyle intervention (Randrianarisoa et al., 2016)

Supplementation with acetyl-L-carnitine (ALC), an acetyl group donor, ameliorates motor and cognitive deficits in other disease models through a variety of mechanisms including altering patterns of histone acetylation resulting in changes in gene expression, and stimulating biosynthetic pathways such as acetylcholine

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