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Yaylaci S, Guler MO, Tekinay AB

13a, Supplementary Table 9), suggesting that the distinct rotamer orientation of H296 ECL2 in Cagri-bound CTR (to accommodate the Glu linker) and/or the observed more open ECL3 conformation induced by the lipidated peptide may facilitate or stabilize an active CTR conformation (Supplementary Fig
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Without these supporting documents, payers may deny authorization under the rationale that oral B12 costs less and should be trialled first-a medically inappropriate request given the intrinsic factor pathophysiology
