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MacMillan CJ, Furlong SJ et al (2012) Bevacizumab diminishes experimental autoimmune encephalomyelitis by inhibiting spinal cord angiogenesis and reducing peripheral T-cell responses

Experimental assays demonstrate that specific XRCC1 variants impair base excision repair efficiency, causally increasing mutation rates upon toxin exposure [273,274,275]
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Consequently, riboflavin may significantly enhance cell longevity by boosting the activity and concentration of antioxidant enzymes, such as superoxide dismutase, catalase, and glutathione peroxidase, that act on deactivating peroxides, which may create an oxidant environment (, )

Table 1 Primers used in various experiments