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GEVALnull was used as an unresponsive negative control

Public comment and physician testimony matter in ongoing PCAC review

J Bone Min Metab

Cell Death Discov 8:29

The suppression of neuroinflammation and reduction of astrocyte-mediated excitatory signaling through MAO-B inhibition highlight its therapeutic potential for chronic pain management, reinforcing the importance of targeting astrocytic MAO-B as a novel strategy in neuropathic pain treatment 540

In contrast, the gdh3 / gdh3 mutant showed a higher concentration of ATP, NAD + , and NADH, but not NADP + or NADPH, when incubated in proline medium when compared to the wild-type strain (Figs 7 and 8)