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28 This is known as restrictive clearance, as only the free, unbound drug is available for metabolism

[DOI] [PubMed] [Google Scholar] 184.Li J, Cao F, Yin H-L, Huang Z-J, Lin Z-T, Mao N, et al

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The amyloid cascade hypothesis which is based on the molecular defects observed in autosomal-dominant EOAD has been severely criticized due to the unsolved paradox: Why do A aggregate into fibrils?, but it is clear that the A sequence, A concentration, and conditions that destabilize A are important factors in aggregation [83, 84]

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