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Studies with Egyptian children and adolescents, for example, show that the null genotype of conferred a 4.2-fold increased risk for the occurrence of DM, and in this case, associations with some biochemical variables and laboratory data were also observed (lipid profile and HbA1c)

(PubMed) de Brito NJ, Rocha ED, de Araujo Silva A, et al

Suzuki, Y., Sato, J., Kawanishi, M

A group of children with ASD and relatively unfavorable redox profiles have been described as having a distinct metabolic endophenotype in previous studies (James et al., Children with ASD who are positive for the binding FRAA did not appear to have significant differences in behavioral, developmental or physiological measures when compared to children with ASD who were negative for the binding FRAA but the group did demonstrate significantly higher serum B12 concentrations

This homogeneity likely supports cellular resilience, ensuring that fluctuations in redox state are uniformly sensed and managed across compartments

doi: 10.1016/j.bbagen.2014.12.001