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In aggregate, whilst it may be premature to speculate, given safety, tolerability and cost effectiveness remain to be confirmed, siRNA inhibition of ANGPTL3 may offer a futuristic panacea for targeting HTG in a wide range of dyslipidaemic settings, including residual elevation in LDL cholesterol in FH [92]

Individual variability in human blood metabolites identifies age-related differences

[10] [13] Currently, the consensus is that the phenotype of spinal muscular atrophy spans a continuum of symptoms without clear delineation of subtypes