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Specifically, dysregulated neuronal energy metabolism can trigger apoptotic cascades via mitochondrial membrane potential collapse or abnormal pyruvate dehydrogenase complex phosphorylation, while targeting monocarboxylate transporter MCT2-mediated lactate shuttling or mitochondrial calcium uniporter (MCU) activity has emerged as a promising therapeutic strategy for neurodegenerative disorders

[PMC free article: PMC2266965] [PubMed: 18073311] 114

Audiol Neurotol 28(1):3242 Prayuenyong P, Baguley DM, Kros CJ, Steyger PS (2021) Preferential cochleotoxicity of cisplatin

Genetic background, environmental and intraluminal factors, and mucosal immune dysregulation have been suggested to be involved in UC pathogenesis [2]

Dual-component gelatinous peptide/reactive oligomer formulations as conduit material and luminal filler for peripheral nerve regeneration

Diabetes Care 2011;34(9):2054-2060