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Mitochondria, the quintessential metabolic organelles, are master regulators of this process

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Variations in the gene can either cause aberrant redox signaling and increase reactive oxygen species, leading to oxidative stress, or lead to higher selenoprotein enzyme levels and reduced oxidative damage, depending on which variation you have

The amount of DEGs under control conditions continued to increase as time passed

Oxidative damage to macromolecules in human Parkinson disease and the rotenone model

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