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Possible mechanisms for the deleterious effects of Hcy on cognitive function are as follows: Hcy leads to DNA breakdown, oxidative damage, and the apoptotic process [7], in addition to as well as excitotoxic cell death by directly activating the neuronal NMDA receptor after formation of homocysteic acid [45]

Prevalence of factor V Leiden in patients with retinal vein occlusion

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Methods 16 , 293300 (2024)

Distinct PRDX isoforms also regulate ferroptosis through signaling functions

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