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Mechanistically, PS-NPs are believed to suppress EAAT2 expression and function indirectly by inducing astrocytic dysfunction, including upregulated glial fibrillary acidic protein expression and heightened oxidative stress

doi: 10.1002/jcla.24430

For instance, a recent study demonstrated that AST suppresses oxidative stress and inflammatory factor production in LPS-induced dendritic cells via the HO-1/Nrf2 pathway [10], enhances defense mechanisms against pathogens, and reduces the incidence of autoimmune diseases

IngoId I, et al

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S.UrajiM.TuyaA.NakamuraY.MurataY