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I provide more context for their work and information about his approach in the final section of this post - its well worth the read

It creeps up on people

Further examination and analysis using the annexin-v binding assay and immunoblotting analysis found that cancer cells with MET amplification are more dependent on the HGF/c-MET signaling pathway for oncogenic growth [42]

ATR inhibitor AZD6738 and WEE1 inhibitor AZD1775 inactivated RAD51-mediated homologous recombination [359], activated cyclin-dependent kinase 1 (CDK1) activity, forced DNA-damaged cells into mitosis, and induced serious mitotic abnormalities and mitotic catastrophes, eventually resulting in TNBC cell death, and improving the sensitivity of TNBC to cisplatin and PARP inhibitors [360, 361]

In addition, as noted above, most item nonresponse was addressed using hot-deck imputation methods and random imputation for a few items when applicable

High Mutational Concordance Between Next Generation Sequencing Profiles of Colorectal Cancer and Pulmonary Metastases
