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Among tyrosine kinase inhibitors, imatinib (a PDGFR inhibitor) and erlotinib (an EGFR inhibitor) induce ROS-dependent apoptosis in melanoma and non-small-cell lung cancer cells, respectively, through disruption of mitochondrial membrane potential upon the stimulation of JNK and p38 phosphorylation 88,89 , while vemurafenib (a BRAF inhibitor) increases the production of superoxide anions with the commensurate depolarization of the mitochondrial membranes in melanoma cells 90

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References 1Peptide bioregulation of aging: results and prospects | Anisimov & Khavinson | 2010 | Biogerontology 2Cerebrolysin for acute ischaemic stroke | Ziganshina, Abakumova & Hoyle | 2020 | Cochrane Database of Systematic Reviews Disclaimer: The above information is for research and educational purposes only and not a substitute for professional medical advice

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All-optical Spatiotemporal mapping of ROS dynamics across mitochondrial microdomains in situ [J]

C., Dedrick, G