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Molecular remedy of complex I defects: rotenone-insensitive internal NADH-quinone oxidoreductase of Saccharomyces cerevisiae mitochondria restores the NADH oxidase activity of complex I-deficient mammalian cells
E., Vazquez, F

doi: 10.3390/blsf2024040040 265 MarraAManousakisVZervasGPKoutisNFinosMAAdamantidiTet al

Asimakopoulou, A., Panopoulos, P., Chasapis, C
Structure, function, and mechanism of ribonucleotide reductases

Tillonen et al conducted a study indicating that this reaction might involve ADH inhibition in the gastrointestinal (GI) tract, instead of in the liver as previously thought
