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Mitochondrial signaling in the vascular endothelium: beyond reactive oxygen species

237 However, the histone marker alteration patterns differ in different stress-induced premature senescence cells based on various stress factors

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They produce a large amount of ROS, which induces damage to the cellular structures (e.g., the damage to DNA, proteins, and lipids) and even the death of the cardiomyocytes, particularly in diabetes [158]

For instance, microbiome-modulating interventions, such as prebiotics that promote butyrate production, may enhance endothelial barrier function

Novel patient-derived models of desmoplastic small round cell tumor confirm a targetable dependency on ERBB signaling