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Under conditions of Na+/K+ ATPase hypofunctionality, sodium accumulates in neurons and alters the resting potential, resulting in altered neuronal excitability [101]

Research models have examined its roles in: angiogenic signaling inflammatory modulation gastrointestinal mucosal protection connective-tissue and ligament studies neural protection models Most findings come from rodent studies and in vitro experiments, where researchers evaluate tissue-specific responses under controlled conditions

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[DOI] [PubMed] [Google Scholar] 273.Linn TC, Pettit FH, Hucho F, Reed LJ

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