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In chemically induced liver injury modelsincluding carbon tetrachloride (CCl 4 ), acetaminophen (APAP), cadmium (Cd), and alcohol (AA) exposureGLPs activate the Nrf2Keap1 antioxidant axis, leading to nuclear translocation of Nrf2 and subsequent upregulation of heme oxygenase-1 (HO-1) and downstream phase II enzymes

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[1] [15] The conserved active site residues in E

To minimize bias, we only included studies of MD that had large sample sizes (25133 children per study

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These results elucidated that the neuroprotective attributes of luteolin are facilitated through the inhibition of endoplasmic reticulum stress in astrocytes, thereby endorsing its prospective application as a therapeutic agent for AD ( in vitro model for AD