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Concurrently with the evolution and establishment of PGHS/PG-dependent mode of NSAID toxicity, a second school of opinions advocating the direct effect of NSAIDs on mitochondria followed by generation of cellular oxidative stress and apoptosis has also strengthened its foothold as an independent pathway of NSAID-induced cytopathology [24]

Riew, T.-R

ii) Database: MEDLINE ( iii) Key words: Insulin-like growth factor-1, IGF-1, tendon injury, tendinopathy, tendon repair iv) Boolean algorithm: ('insulin-like growth factor-1' OR 'IGF-1') AND ('tendon' OR 'tendinopathy' OR 'tendon repair')

The impact of oxidation-reduction potential in follicular fluid on intracytoplasmic sperm injection outcomes

Ianya selamat
