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Description
Further research confirms that ACSL4 suppression in early ischemic stages is time-dependent and likely driven by increased HIF-1, which binds to the ACSL4 promoter and suppresses its expression (137)

doi: 10.1016/j.prp.2024.155553 47 SangkhaeVNemethE

Brain Res Mol Brain Res

It is highly valued for its performance-enhancing and regenerative properties

It protects cells from damage caused by harmful molecules known as free radicals

Pro-Resolving Lipid Mediators in Sepsis and Critical Illness
