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Notably, breast cancer cells achieve metabolic adaptation by hijacking the autophagy mechanism: under glucose-deprivation conditions, MANF-mediated mitophagy eliminates dysfunctional mitochondria through the PRKN/Parkin-dependent pathway, maintains the ATP supply and promotes tumour survival [112], suggesting that targeting the MANFPRKN interaction can break the drug resistance barrier of breast cancer under metabolic stress

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White, MG, Damania, A, Alshenaifi, J, Sahasrabhojane, P, Peacock, O, Losh, J, Wong, M, Berkova, Z, Nguyen, S, Chang, GJ, Wargo, J, Ajami, NJ, Kopetz, S , You, YN

Graphical Abstract Access this article Similar content being viewed by others Data Availability No datasets were generated or analysed during the current study

The same programmed deficits in muscle structure and function are found in numerous neurodegenerative syndromes and disease-related cachexia 3

has served on scientific advisory boards at Merck Shape and Dome, Astra Zeneca, Novo Nordisk and Otsuka