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However, since oxidative damage is a known mechanism for secondary degeneration in acute spinal cord injury, administration of antioxidants during the first hours or days after insult has been shown to stabilize mitochondrial bioenergetics, spare nerve tissue and, in some cases, result in modest neurological recovery [5], [11][15]
Numerous later studies have shown the association between increased plasma TMAO levels with a higher risk of cardiovascular events [63,64,65,66]
Biochem Biophys Res Commun
Those experiencing headaches with fatigue, light or sound sensitivity
