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So why could protein oxidation, in particular, reversible oxidation-induced by ischemic tolerance be involved in protection against subsequent ischemic injury

although their phagocytic activity and release of neutrophil extracellular traps (NETs) contribute to pathogen clearance, excessive activation and dysregulated NET formation cause direct tissue injury (Long et al., 2024)

Of note, through histopathological and immunofluorescence analyses of immune cell subsets, we confirmed that lowering oxalate via AGXT overexpression or pharmacological inhibition of LDHA and GO reduces steatohepatitis aligned with the decrease in distinct subsets of recruited monocyte-derived macrophages (CCR2 + and TREM2 + macrophages) that are known to infiltrate the liver in MASH and form crown-like structures surrounding hepatocytes with large lipid droplets 49

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Cel Mol Immunol 18 (1), 4556

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