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In knock-in mice expressing DOR-eGFP, a challenge with SNC-80 but not ARM-390 induces a tolerance to analgesic response in a model of inflammatory pain with a concomitant G protein uncoupling in both brain and spinal cord homogenates (Pradhan et al., 35 S]GTPS binding was reduced compared to naive animals (Bohn et al., Phosphorylation Anti-nociceptive tolerance induced by morphine, meperidine, and fentanyl was shown to be reduced by PKC inhibitors while DAMGO-induced tolerance and MOR desensitization was shown to rely on GRK (Hull et al., in vitro experiments showed that S375 is phosphorylated by GRK5 upon morphine exposure (Doll et al., 2004), S375A knock-in mice still present anti-nociceptive tolerance upon acute and chronic exposure to morphine (Grecksch et al., 2004

295 In addition, high expression of TLR4 can increase H 2 O 2 production in the colonic mucosa of UC mice, induce redox activity in the mucosa, exacerbate oxidative stress, and promote the disease progression of UC