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The studies performed in subjects with chronic exposure to arsenic [27], or with clinically relevant intoxication by mercury [76], and in experimental animals intoxicated by mercury [77], cadmium and lead [78] show that the levels of some indicators of oxidative stress are displaced towards a pro-oxidative condition

Also, the 50% protective level that was estimated each time a study was left out of the analysis provides a metric of the sensitivity of the model to the inclusion of each study

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Moreover, many of them must deal with the scars that come after

Ozone exposure induces respiratory barrier biphasic injury and inflammation controlled by IL-33

D.: 1.0% max
