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It interacts with A peptides, preventing their oligomerization and neurotoxic cascade

In contrast, the (-)-Vestitol group ( P = 0.0121

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Since cysteine is known to be neurotoxic in excessive amounts [32], treatment with N-acetylcysteine should not be recommended for patients with glutathione synthetase deficiency as this may increase the intracellular cysteine levels even more

Current work is directed toward understanding the molecular mechanism by which sulfenylation of EGFR Cys797 enhances kinase activity