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This may also explain the loss of contractile function during metabolic inhibition (Figures 1, 2), where the Kir6.2/SUR2A channels, the well-established canonical cardiac K ATP channel subunits, would also be open (Brennan et al., 2024

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This finding is particularly noteworthy because it not only emphasizes the role of lifestyle factors in the pathogenesis of glaucoma but also points to a potential point of intervention to prevent or delay disease progression by modifying dietary sodium intake (99)