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GHK-Cu's 30-minute plasma half-life is pharmacologically irrelevant to its dosing schedule because the peptide's mechanism is not receptor occupancy

Yet the cognitive enhancement effects triggered during exposure persist for weeks through sustained upregulation of hepatocyte growth factor (HGF) and its receptor c-Met in hippocampal tissue

Decreased levels of proapoptotic factors and increased key regulators of mitochondrial biogenesis constitute new potential beneficial features of long-lived growth hormone receptor gene-disrupted mice