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doi: 10.1152/physiolgenomics.00016.2023 88
This indicates an intramolecular amidation subsequent to thiolysis ( Figure 3A )
Of note, while catagen induction in wild-type mice in this particular genetic background strictly occurs at P18, catagen induction in the double mutant substantially varies
Disclaimer: This article is for informational purposes only and does not constitute medical advice
Implications of ferroptosis in disease Excessive or defective ferroptosis can contribute to pathological cell loss, as well as to malignant processes
