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These mechanisms elucidate the pathological underpinnings of ferroptosis across a spectrum of diseases and suggest potential therapeutic targets

Additionally, O 2 can react with nitric oxide (NO) to generate excessive ONOO, activating caspases, causing lipid peroxidation, and leading to either apoptosis or necrosis [49]

We follow [30, 31] and use a reversible bi-reactant Michaelis-Menten mechanism

Interactions of -L-glutamyltaurine with excitatory aminoacidergic neurotransmission

Anti-stress effect of theanine on students during pharmacy practice: positive correlation among salivary -amylase activity, trait anxiety and subjective stress