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GPX2 inversely correlated with expression of multiple pro- inflammatory cytokines/chemokines and NF-kB activation in cell lines and knockdown of GPX2 led to increased secretion of prostaglandin E2 (PGE2) and interleukin-6

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Research models suggest that it interacts with pathways involved in tissue repair coordination rather than functioning as a hormone or growth factor

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System xc was recently described as the most upstream node in a novel form of regulated necrotic cell death, called ferroptosis