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In AD and PD, the failure of glial cells to degrade pathogenic aggregates, such as A, tau, and -syn, precipitates chronic inflammation and secondary neurotoxicity
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Song R-H, Wang B, Yao Q-M, Li Q, Jia X, Zhang JA
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endoplasmic reticulum for re-esterification
For instance, it could be speculated that such discrepancies may reflect compensatory mechanisms aimed at correcting underlying vulnerability, consistently with previous findings [49] showing an increase in C3 during antidepressant treatment
