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Notably, although both CCKRs adopt the canonical seven-transmembrane architecture, they differ from other reported class-A GPCRs with a shortened TM4 helix, which is attributed to the unwinding of the helix at the extracellular half caused by the presence of P 4.59 and the additional P 4.61 (the superscripts refer to the BallesterosWeinstein number) 44

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In both conditions, the stimulation of the exchange protein directly activated by cAMP/Ras-related protein/phospholipase C signaling triggered a series of transcriptional events that ultimately led to Adrb3 gene repression