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Together with reninangiotensinaldosterone activation, endothelin-1 (ET-1) upregulation, and smooth-muscle hypertrophy in resistance vessels, these changes contribute to the hypertensive phenotype in MetS and T2DM [1001,1002,1003,1004,1005,1006]

People with certain cancers, particularly hematologic (blood-related) or liver malignancies 11

We postulate that boosting mitochondrial metabolism might be a potential therapeutic strategy to induce antidepressant effects

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Additionally, GPX4 enhances the activity of muscle/ER Ca 2+ -ATPase (SERCA), maintaining calcium homeostasis and excitation-contraction coupling, thereby alleviating sarcopenia progression (72)